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Frontiers | The Role of Microglia and Astrocytes in Huntington's Disease

One important consideration is that any virulence determinant should fulfill molecular Koch’s postulates (Falkow, 1988), which essentially require that (i) a phenotype or gene be specific to pathogenic members of a genus, (ii) inactivation of a gene results in mutant with reduced infectivity (the ability to cause infection) or pathogenicity (the ability to cause disease), and (iii) complementation of the mutant restores the properties of the wild-type

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Namely, given together (L-NAME+L-arginine) regularly attenuated or antagonized each other's response